Fig. 3
From: Diabetes mellitus in a patient with glycogen storage disease type Ia: a case report
Alternative pathways for glucose-6 phosphate leads to lactic acidosis, hyperuricemia, and hyperlipidemia: In glycogen storage disease type 1a, there is deficient glucose-6-phosphatase, which results in the shunting of the substrate glucose-6-phosphate away from glucose, into alternative pathways. This results in elevated lactic acid, hyperuricemia, and hyperlipidemia. Source: Froissart R, et al. [3]